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While their primary indications and mechanisms may vary (Figure 5), their ability to favorably influence the redox balance, as supported by the clinical studies on biomarkers and surrogate endpoints listed in Table 1, contributes to their overall beneficial effects

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Suppression of GSTP at the transcript level induced oxidative stress based on mechanisms that intervened in mitochondrial homeostasis, which might be more rapidly and strongly than suppressing the expressed GSTP protein

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